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The role of oxidative stress in sleep deprivation-induced reproductive dysfunction: A rationale for anti-oxidant intervention April 20, 2026
Sleep deprivation (SD) is a significant risk factor for reproductive dysfunction. This review aims to synthesize evidence establishing that oxidative stress (OS) is the key pathological mediator of SD-induced reproductive impairments in both sexes and to provide a comprehensive rationale for anti-oxidant intervention. We conducted a narrative review of peer-reviewed studies (2000-2025) from PubMed, ScienceDirect, […]
Protective effect of wild polysaccharides extracted from Ulva prolifera on oxidative stress damage in valproic acid-induced neuronal cells April 20, 2026
CONCLUSIONS: PUPs demonstrate a significant protective effect against VPA-induced oxidative stress damage in HT22 neuronal cells. As a natural antioxidant, PUPs hold promising potential for the prevention and adjunct treatment of ASD-related neuronal oxidative stress damage.
Enhanced bone regeneration and cellular protection from oxidative stress using a vitamin C-based scaffold April 20, 2026
CONCLUSION: The VitC@HAP-HYA composite effectively combines multifunctional qualities, demonstrating superior bioactivity, increased osteogenic potential, and vital cytoprotective features. These results robustly endorse its potential as a sophisticated framework for bone tissue engineering, including applications such as anti-cancer bone excision repair.
Modulation of dental caries-associated oxidative stress by different restorative materials: A salivary malondialdehyde-based in vivo study April 20, 2026
CONCLUSION: It may, therefore, be concluded that Cention N restorative material results in rapid and superior reduction of caries-induced inflammatory stress in the body as compared to nano-composite restorative material.
Recent advances in tendon redox biology: the interplay of oxidative stress, calcium signaling, and antioxidant defence mechanisms April 20, 2026
Tendon injuries are increasingly recognized as conditions driven not only by mechanical overload but also by complex molecular imbalances, particularly involving oxidative stress. Recent evidence highlights the central role of reactive oxygen species (ROS), originating primarily from mitochondrial respiration and NADPH oxidase activation, in regulating cellular responses during tendon injury and repair. Mechanical loading and […]